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Focal ischemia followed by chronic hypoperfusion triggers changes in AQP4 distribution from perivascular to parenchymal and increases amyloid- deposits which contributes to PSCI ( Permeability of the scar-border allows the accumulation of antibodies in the parenchyma after stroke (Zbesko et al., 2018), which is linked to the development of PSCI ( Neuroinflammation and oxidative stress linked to secondary neurodegeneration after ischemic stroke is proposed to drive the development of PSCI

Rank of proteoglycans by fold change in gene expression in ccRCC tumours relative to paired adjacent kidney

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